Knee Dislocation

Aka: Knee Dislocation, Tibial Femoral Dislocation, Knee Subluxation, Multiligament Knee Injury

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II. Background

  1. Uncommon, but potentially limb threatening injury

III. Pathophysiology

  1. High energy knee injuries result in shearing forces that tear multiple ligaments
  2. Knee Dislocation requires the disruption of 2-3 of the four major knee ligaments
    1. Anterior Cruciate Ligament Tear
    2. Posterior Cruciate Ligament Tear
    3. Knee Medial Collateral Ligament Tear
    4. Knee Lateral Collateral Ligament Tear
  3. Instability results from multiple ligamentous injuries
    1. Allows for translation of tibia and fibula in relation to the femur
  4. Associated Popliteal Artery Injury
    1. Popliteal artery is tethered above (tendinous hiatus) and below (soleus tendinous arch) the popliteal fossa
    2. Dislocation stretches the popliteal artery which has decreased flexibility due to its tethering

IV. Causes

  1. Motor Vehicle Accident (two thirds of cases)
  2. Collision Sports (e.g. football, rugby, soccer)
  3. Fall from height
  4. Downhill Skiing
  5. Cutting sports
    1. Knee Dislocation may occur with lower energy if there is rotation prior to shearing forces
    2. Athlete attempts to change direction with a planted lower extremity
      1. Results in knee hyperextension
  6. Severe Obesity
    1. Single condition in which Knee Dislocation may occur even without high velocity Trauma
    2. Obesity also complicates the dislocation diagnosis (obscured landmarks) and vascular monitoring
    3. Typically requires slight rotation of the knee prior to experiencing shearing forces
      1. Change in direction during ambulation
    4. Multiple case studies demonstrating low mechanism dislocations, typically in BMI >40
      1. Popliteal artery injury in 40%
      2. Peroneal nerve injury in 40%
    5. High risk of amputation (as high as 20% of Knee Dislocations)
      1. Ankle brachial index (and CTA if <0.9)
    6. References
      1. Azar (2011) Am J Sports Med 39(10): 2170-4 [PubMed]
      2. Folt (2012) Am J Emerg Med 30(9): 2090.e5-2090.e6 [PubMed]
      3. Georgiadis (2013) J Vasc Surg 57(5): 1196-203 [PubMed]
  7. Total Knee Arthroplasty (TKA)
    1. Anterior dislocation is more common than posterior dislocation
    2. Prevalence of Knee Dislocation after TKA: 0.15 to 0.5%
    3. Causes
      1. Implant malposition
      2. Flexion-Extension gap mismatch
      3. Excessive soft tissue release or laxity
      4. Incorrect primary implant selection
    4. Emergent Management is the same for native Knee Dislocation (see below)
      1. Emergent knee relocation to reduce the risk of neurovascular compromise
      2. Neurovascular exam and evaluation including ABI or CTA as indicated (see below)
      3. Revision of TKA
    5. References
      1. Rashidzada (2020) Crit Dec Emerg Med 34(12): 12-3

V. Types

  1. Most common types
    1. Anterior Dislocation (40% of cases, most common)
      1. Severe Knee hyperextension injury (>30 degrees with torn posterior stabilizing elements)
    2. Posterior Dislocation
      1. Direct blow to the proximal tibia (e.g. knee strikes car dashboard in MVA)
  2. Less common types
    1. Medial dislocation
      1. Results from valgus force
    2. Lateral Dislocation
      1. Results from varus force
    3. Rotary Dislocation
      1. Planted foot with counter rotation of the body
      2. May result in a posterolateral dislocation (non-reducible without surgery)

VI. Exam

  1. See Knee Exam
  2. Perform before and after reduction
  3. Observation
    1. Knee Effusion or swelling, Ecchymosis or deformity
  4. Palpation
    1. Focal tenderness over joint line and bony prominences
  5. Range of motion
    1. Passive and active range of motion if patient is able
  6. Multidirectional instability
    1. Multidirectional instability may be only finding if knee spontaneously reduced prior to presentation
    2. At least 2-3 of four ligaments are likely injured (ACL Tear, PCL Tear, LCL tear, MCL tear)
      1. Evaluate each knee ligament
  7. Vascular exam (especially popliteal artery distribution)
    1. Perfusion Assessment
      1. Dorsalis pedis pulse
      2. Posterior tibial pulse
      3. Capillary Refill
      4. Ankle-Brachial Index (ABI)
        1. Normal ABI (as well as pulses, Capillary Refill) may not exclude popliteal artery disruption
        2. Ankle-Brachial Index (ABI) < 0.9 requires advanced imaging
    2. Hard signs of vascular injury
      1. Distal pulse loss or ischemia (e.g. pallor, coolness)
      2. Active bleeding
      3. Expanding Hematoma
      4. Palpable thrill or bruit over popliteal artery
      5. Paresthesias or paralysis
  8. Neurologic Exam (especially peroneal nerve)
    1. First web space and dorsal foot Sensation
    2. Ankle dorsiflexion
  9. Skin changes
    1. Dimple Sign
      1. Anteromedial skinfold at medial joint line
      2. Seen in posterolateral dislocation (not reducible without surgery)
    2. Skin necrosis
      1. Entrapped skin at femoral condyle
    3. Overlying Laceration
      1. Suggests open Knee Dislocation (accompanies 20-30% of Knee Dislocations)

VII. Imaging

  1. Ankle-Brachial Index (ABI) under exam as above
    1. Normal ABI (as well as pulses, Capillary Refill) may not exclude popliteal artery disruption
    2. Ankle-Brachial Index (ABI) < 0.9 requires advanced imaging
  2. Knee XRay
    1. Pre-reduction
      1. Do not delay reduction for imaging if any signs of vascular compromise
      2. XRay evaluates for Fracture
    2. Post-reduction XRay
      1. Associated lower extremity Fracture in almost 60% of Knee Dislocations
      2. More common Fractures include Tibial Plateau Fracture, tendon avulsion Fracture at lateral tibial condyle
  3. Vascular Ultrasound
    1. Arterial Doppler Ultrasound
  4. CT Angiogram (or MR Angiogram)
    1. Indicated post-reduction if signs of vascular compromise (popliteal artery disruption)
    2. Critical Limb Ischemia requires emergent vascular surgery without delay of angiogram (see below)

VIII. Precautions

  1. Knee Dislocation is a surgical emergency requiring immediate reduction
    1. Delay in reduction risks limb-threatening vascular compromise
    2. Pre-reduction imaging is only indicated if no signs of vascular compromise
  2. Knee spontaneous reduction prior to presentation occurs in >50% of patients
    1. Do not dismiss patient report of "popping out-and-in Sensation" simply as Patella subluxation
    2. Multidirectional instability may be only finding if knee spontaneously reduced prior to presentation
    3. Knee Dislocation may occur with low energy mechanism and confers much higher risk than Patella subluxation
    4. Follow the same vascular evaluation and monitoring if Knee Dislocation is suspected
  3. Have a high index of suspicion for vascular injury (popliteal artery injury in up to 20-40% of patients)
    1. Popliteal artery repair delayed >8 hours is associated with Limb Amputation in 85% of patients
      1. (2007) J Trauma 63(4): 855-8 [PubMed]
    2. Vascular injury may be present in up to 9% of cases despite normal distal pulses (high risk of amputation)
      1. Confirm with ABI and Doppler Ultrasound
      2. Obtain CT Angiogram if Ankle-Brachial Index <0.9
    3. More than 50% of Knee Dislocations spontaneously relocate prior to emergency department presentation
      1. Follow post-reduction plan below

IX. Management: Knee Joint Reduction

  1. Perform knee reduction without delay
  2. Procedural Sedation
  3. Technique (requires 2 providers)
    1. One provider grasps the distal femur to stabilize
    2. Other provider grasps the proximal tibia
      1. Avoid compression at the popliteal fossa (popliteal artery)
      2. Apply longitudinal traction to tibia
      3. Reverse the dislocation if traction alone does not reduce the dislocation
        1. Continue longitudinal traction
        2. Push the tibia posteriorly in anterior dislocation
        3. Pull the tibia anteriorly in posterior dislocation
  4. Splint knee after reduction
    1. Apply Knee Immobilizer or long leg splint
    2. Prevents repeat dislocation in the acute period
    3. Splint or Knee Immobilizer in 15 to 20 degrees flexion
  5. Failed reduction
    1. Emergent orthopedic Consultation
    2. Posterolateral dislocations are not reducible without surgery
  6. Emergent Surgical Intervention Indications
    1. Prolonged ischemia or other hard signs of vascular injury (see exam above)
    2. Knee reduction fails or cannot be maintained
    3. Posterolateral dislocation (requires both emergent orthopedic consult and vascular consult)
    4. Open Knee Dislocation

X. Management: Evaluate for vascular compromise (popliteal artery injury)

  1. No vascular injury
    1. Findings
      1. Strong dorsalis pedis pulse and posterior tibial pulse
      2. Ankle-Brachial Index >0.9
      3. Bedside arterial duplex Ultrasound normal
    2. Management
      1. Observation hospital stay
      2. Serial distal vascular examinations
      3. Observe for distal extremity paleness, Paresthesias, paralysis
      4. Orthopedic Consultation for reconstructive surgery
  2. Possible vascular injury
    1. Findings
      1. Adequate limb perfusion despite abnormal vascular findings
      2. Asymmetric dorsalis pedis pulse or posterior tibial pulse
      3. Ankle-Brachial Index <0.9
      4. Bedside duplex Ultrasound abnormal
    2. Management
      1. Urgent vascular surgery Consultation
      2. Urgent CT angiogram (or other angiographic assessment of popliteal artery)
  3. Vascular compromise (limb-threatening)
    1. Findings
      1. Weak or absent dorsalis pedis pulse and posterior tibial pulse
      2. Signs of limb ischemia or vascular injury
    2. Management
      1. Emergent vascular surgery Consultation for immediate repair

XI. Complications

  1. Popliteal artery injury (32-40% of Knee Dislocations)
    1. Results in limb threatening vascular injury
    2. Popliteal vein injury may also occur
    3. Devascularization results in nearly 100% amputation rate at 8 hours
  2. Peroneal nerve injury (14-35% of Knee Dislocations)
    1. Results in loss of first web space and dorsal foot Sensation
    2. Results in loss of ankle dorsiflexion and toe extension
  3. Lower leg Compartment Syndrome
    1. Late finding if due to popliteal artery injury
  4. Cruciate Ligament Teat (ACL, PCL)
  5. Deep Vein Thrombosis
  6. Tibial Plateau Fracture
  7. Tibial shaft Fracture
  8. Fibular Head Fracture
  9. Severe knee ligamentous disruption

XII. References

  1. Grozenski and Kiel (2019) Crit Dec Emerg Med 33(10):10-1
  2. Kirwin, Conroy, McGrath (2021) Crit Dec Emerg Med 35(7): 15-24
  3. Mason and Campagne in Herbert (2018) EM:Rap 18(3):12-3
  4. Bachman in Wolfson and Stack (2014) Knee Dislocation and Reduction, UpToDate, accessed 6/5/2014
    1. http://www.uptodate.com/contents/knee-tibiofemoral-dislocation-and-reduction
  5. Kelleher in Brenner (2013) Knee Dislocation, Medscape Emedicine, accessed 6/5/2014
    1. http://emedicine.medscape.com/article/823589-clinical#showall

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