II. Epidemiology
- Ages 30 to 60 years old
- No significant gender predisposition
- Uncommon: 0.5 cases per 100,000 person years
III. Pathophysiology
- Longus coli is one of 4 cervical flexor Muscles
- Calcific Tendonitis develops within the longus coli Muscle tendons
- Superior aspect tendons are most susceptible (C1 to C3, down to C5 region)
- Calcium deposits within tendon as a response to decreased local strength
- Tendon rupture results in release of hydroxyapatite crystals into soft tissue
- Local crystal release results in aseptic inflammatory response
- Precipitating injuries
- Repetitive Trauma with secondary localized tendon ischemia and necrosis
- Course
- Resolves spontaneously over weeks
- Resolves as hydroxyapatite crystals are phagocytosed by Macrophages
IV. Symptoms
- Acute Neck Pain
- Neck stiffness and decreased range of motion
- Dysphagia
- Odynophagia
- Globus Hystericus
- Low grade fever
V. Labs
- Leukocytosis (mild)
- Inflammatory markers (e.g. CRP) mildly increased
VI. Imaging
- CT Cervical Spine
- Gold standard for identifying specific calcification changes of ACTLC
- Calciium deposition within the longus colli
- Prevertebral edema
- Rim enhancement is absent in ACTLC (but present in abscess)
VII. Differential Diagnosis
VIII. Management
- Conservative management
- Resolves over the course of weeks spontaneously
- No indication for extracorporeal shock wave therapy or surgery
- Analgesics and antiinflammatory agents